SARS-CoV-2 spike and nucleocapsid proteins fail to activate human dendritic cells or γδ T cells
Frontiers Dok3 restrains neutrophil production of calprotectin during TLR4 sensing of SARS-CoV-2 spike protein
SARS-CoV-2 Spike protein induces TLR4-mediated long-term cognitive dysfunction recapitulating post-COVID-19 syndrome in mice - ScienceDirect
SARS-CoV-2 suppresses TLR4-induced immunity by dendritic cells via C-type lectin receptor DC-SIGN
SARS-CoV-2 spike protein induces a differential monocyte activation that may contribute to age bias in COVID-19 severity
SARS-CoV-2 spike and nucleocapsid proteins fail to activate human dendritic cells or γδ T cells
SARS-CoV-2 activates the TLR4/MyD88 pathway in human macrophages: A possible correlation with strong pro-inflammatory responses in severe COVID-19 - ScienceDirect
Differential Expression Patterns of Toll-like Receptors in COVID-19 Patients
Frontiers Toll-Like Receptor Signaling in Severe Acute Respiratory Syndrome Coronavirus 2-Induced Innate Immune Responses and the Potential Application Value of Toll-Like Receptor Immunomodulators in Patients With Coronavirus Disease 2019
Selectively expressing SARS-CoV-2 Spike protein S1 subunit in cardiomyocytes induces cardiac hypertrophy in mice
SARS-CoV-2 nucleocapsid protein, rather than spike protein, triggers a cytokine storm originating from lung epithelial cells in patients with COVID-19
SARS-CoV-2 spike protein induces the cytokine release syndrome by stimulating T cells to produce more IL-2
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SARS-CoV-2 spike protein induces the cytokine release syndrome by stimulating T cells to produce more IL-2
