PDF) Arntl deficiency in myeloid cells reduces neutrophil recruitment and delays skeletal muscle repair
Regulation of skeletal muscle regeneration by CCR2-activating chemokines is directly related to macrophage recruitment
ROS-activated CXCR2+ neutrophils recruited by CXCL1 delay denervated skeletal muscle atrophy and undergo P53-mediated apoptosis
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Regulatory interactions between muscle and the immune system during muscle regeneration
Bmal1 regulates inflammatory responses in macrophages by modulating enhancer RNA transcription
Macrophages recruited via CCR2 produce insulin-like growth factor-1 to repair acute skeletal muscle injury. - Abstract - Europe PMC
Cells, Free Full-Text
Frontiers Inflammatory myopathies and beyond: The dual role of neutrophils in muscle damage and regeneration
Neutrophils as regulators of cardiovascular inflammation
Plasminogen system. Plasminogen is the inactive substrate of the
Immunohistochemistry of macrophages 2 (expressing mannose receptor) in
Frontiers A Tissue-Specific Rhythmic Recruitment Pattern of Leukocyte Subsets
PDF] Impaired ECM Remodeling and Macrophage Activity Define Necrosis and Regeneration Following Damage in Aged Skeletal Muscle
