Obesity-linked suppression of membrane-bound O-acyltransferase 7 (MBOAT7) drives non-alcoholic fatty liver disease
From sugar to liver fat and public health: systems biology driven studies in understanding non-alcoholic fatty liver disease pathogenesis, Proceedings of the Nutrition Society
The diagnostic conundrum in non-alcoholic fatty liver disease
PDF) Genetic risk factors associated with NAFLD Hepatoma Research and DongYun Kim
Genetic and metabolic aspects of non-alcoholic fatty liver disease (NAFLD) pathogenicity, Egyptian Journal of Medical Human Genetics
PGHN :: Pediatric Gastroenterology, Hepatology & Nutrition
Precision medicine in nonalcoholic fatty liver disease: New therapeutic insights from genetics and systems biology
Obesity-linked suppression of membrane-bound O-acyltransferase 7 (MBOAT7) drives non-alcoholic fatty liver disease
Enhancing Hepatic MBOAT7 Expression Does Not Improve Nonalcoholic Steatohepatitis in Mice
Frontiers MBOAT7-TMC4 rs641738 Is Not Associated With the Risk of Hepatocellular Carcinoma or Persistent Hepatitis B Infection
Hepatocellular carcinoma & nonalcoholic fatty liver disease
Lack of evidence supporting a role of TMC4-rs641738 missense variant—MBOAT7- intergenic downstream variant—in the Susceptibility to Nonalcoholic Fatty Liver Disease
The MBOAT7-TMC4 Variant rs641738 Increases Risk of Nonalcoholic Fatty Liver Disease in Individuals of European Descent - ScienceDirect
